关键词： 普济痔疮栓   痔病   内痔   肛窦炎   综述  

摘要： 针对内痔出血、混合痔、肛窦炎、便秘等疾病,普济痔疮栓既可单独使用,也可与其他药物或者其他治疗方法联合应用。普济痔疮栓在肛肠外科应用广泛,临床疗效显著,不良反应少,患者可以耐受,适用人群广。

关键词： 幽门螺杆菌   感染   胃肠道疾病   相关性  

摘要： 探讨分析消化内科患者幽门螺杆菌感染现状以及其对胃肠疾病的相关性。方法:对2018年8月至2019年9月在我院收治的消化内科患者共200例进行研究。研究了85名幽门螺杆菌感染患者。他们的临床资料与年龄、疾病类型、有无吸烟饮酒的分布、胃肠道感染的因素也进行了分析比较。结果:患者的患病率为42.5%,患者的年龄、职业、饮酒吸烟、胃肠道疾病等均为可能感染幽门螺杆菌的因素,复合性溃疡、十二指肠溃疡等幽门螺杆菌感染率高于其他消化道疾病。结论:对消化内科患者幽门螺杆菌感染的筛查及与消化道疾病的关系分析探讨,可有效预防幽门螺杆菌的发生,降低患者感染率。

关键词： 《五十二病方》   胃肠疾病   升阳   暑湿   足浴   中医外治方法   外洗   固脱  

摘要： 春天洗脚,升阳固脱;夏天洗脚,暑湿可祛;秋天洗脚,肺润肠濡;冬天洗脚,丹田温灼。天气转冷,很多胃肠疾病患者问起冬季是否可以足浴?足浴能改善胃肠疾病吗?用什么药好?当然可以足浴。足浴是传统中医外治方法的一种,具有悠久的历史。早在先秦时期的《五十二病方》中就提出“温熨”“药摩”“外洗”,这也是足浴的前身。

关键词： 图木舒克市   幽门螺杆菌感染率   胃十二指肠疾病   分布特征  

摘要： 探究新疆生产建设兵团第三师图木舒克市地区人群中总体幽门螺杆菌(HP)感染得相关情况与其在胃十二指肠疾病得分布特征。方法:对2016年1月至2017年7月间在舒克市人民医院因胃十二指肠疾病进行尿素酶抗体检测和胃镜检查的628例受检者幽门螺杆菌(HP)感染情况进行统计分析,根据受检者的性别、民族、年龄、胃镜诊断等相关影响因素的HP感染率与胃十二指肠疾病分布特征。结果:经对受检者尿素酶抗体检测观察Hp感染情况发现,本次628例受检者中HP阳性感染率为50.6%(318/628例);胃镜诊断分析可见,维吾尔族受检者的HP阳性感染率明显高于汉族受检者,以上比较有统计学意义(P<0.05);经检验提示HP感染与年龄、性别无明显相关,年龄组间感染率比较无统计学意义(P>0.05),60岁以下人群有胃炎患病率更高(P<0.05);胃镜诊断与受检者的HP感染无明确关联,(P>0.05)。结论:图木舒克市人民医院胃镜受检者总的HP感染率为50.6%,受检者HP感染率与民族关联性较强,与性别、年龄、胃镜诊断等其他因素无明确关系,受检中胃炎受检者的HP感染检出率更高。

关键词： 灌水疗法   猪养殖   猪胃肠病  

摘要： 生猪在养殖过程中,需要注意疾病的防制,如果猪发生胃肠问题,可以选用灌水疗法进行治疗。本文首先对灌水疗法的适用症状加以阐述,再对灌水疗法的优势进行介绍,最后对具体的治疗方法和配药配方予以探究。

关键词： 肛肠疾病   手术   右美托咪啶   麻醉  

摘要： 目的:观察右美托咪啶在肛肠疾病患者麻醉中的应用效果。方法:将150例肛肠疾病手术患者随机分为观察组和对照组,每组75例。所有患者均行腰麻-硬膜外联合麻醉,对照组患者使用罗哌卡因进行麻醉,观察组在对照组的基础上持续静脉注射右美托咪啶进行麻醉,对照组患者注射与右美托咪啶等量的生理盐水,观察2组患者神经阻滞情况、运动阻滞情况、生命体征监测、术后镇痛效果及并发症情况。结果:观察组患者神经阻滞起效时间短于对照组(P <0.05),持续时间长于对照组(P <0.05);观察组中运动阻滞评为1分者较对照组少,评为2、3分者多于对照组(P <0.05);麻醉前、麻醉30min后患者生命体征指标组间、组内比较差异无统计学意义(P>0.05);术后观察组患者疼痛评分明显低于对照组(P <0.05);2组患者并发症发生率比较差异无统计学意义(P>0.05)。结论:右美托咪啶用于肛肠疾病患者手术中能够获得更好的麻醉效果,可有效减轻患者术后疼痛,且临床用药安全性较高。

关键词： granular analyses evaluating other IBD medication classes   interval   IBD   inflammatory bowel disease   SECURE-IBD   Surveillance  

关键词： child   Crohn disease   inflammatory bowel disease   oral disease   oral manifestation   pediatric   ulcerative colitis  

摘要： Inflammatory bowel diseases (IBD) represent a group of chronic inflammatory disorders of the gastrointestinal tract that lead to impaired quality of life and substantial health care costs. Up to 50% of pediatric IBD cases present with manifestations in the oral cavity. These may develop in nearly every oral tissue, including the soft tissues, tongue, lips, teeth, and lymph nodes. The goal of this review is to offer a systematic approach to diagnose and manage commonly encountered oral manifestations of pediatric IBD. This knowledge is critical for enhancing the comprehensive care and quality of life of children with these debilitating diseases.

关键词： Inflammatory bowel diseases   Risk factors   Oral microbiota dysbiosis   Ectopic colonization   Intestinal epithelial barrier   Microbial-based treatment strategies  

摘要： Inflammatory bowel disease (IBD) is a group of chronic intestinal inflammatory disorders with a prolonged duration characterized by recurrent relapse and remission. The exact etiology of IBD remains poorly understood despite the identification of relevant risk factors, including individual genetic susceptibility, environmental triggers, and disruption of immune homeostasis. Dysbiosis of the gut microbiota is believed to exacerbate the progression of IBD. Recently, increasing evidence has also linked oral microbiota dysbiosis with the development of IBD. On the one hand, IBD patients show significantly unbalanced composition and function of the oral microbiota known as dysbiosis. On the other, overabundances of oral commensal bacteria with opportunistic pathogenicity have been found in the gut microbiota of IBD patients. Herein, we review the current information on the causative factors of IBD, especially recent evidence of IBD-associated oral microbiota dysbiosis, which has seldom been covered in the previous literature review, highlighting the pathogenic mechanisms of specific oral bacteria in the development of IBD. Ectopic colonization of several oral bacteria, including a subset of Porphyromonas gingivalis, Streptococcus mutans, Fusobacterium nucleatum, Campylobacter concisus, and Klebsiella pneumoniae, may lead to destruction of the intestinal epithelial barrier, excessive secretion of inflammatory cytokines, disruption of the host immune system, and dysbiosis of gut microbiota, consequently aggravating chronic intestinal inflammation. Studying oral microbiota dysbiosis may open future horizons for understanding IBD pathogenesis and provide novel biomarkers for IBD. This review also presents the current treatment and new perspectives for IBD treatment.